Bronnen

Een overzicht van gebruikte bronnen.

E-books

  • Chen, Xi, et al. “Chronic physiologic hyperglycemia impairs insulin-mediated suppression of plasma glucagon concentration in healthy humans.” Metabolism 142 (2023): 155512.

Aanhoudende fysiologische hyperglykemie (hoge bloedsuikerspiegel) veroorzaakt insulineresistentie in het hele lichaam.

  • Giri, Biplab, et al. “Chronic hyperglycemia mediated physiological alteration and metabolic distortion leads to organ dysfunction, infection, cancer progression and other pathophysiological consequences: an update on glucose toxicity.” Biomedicine & pharmacotherapy 107 (2018): 306-328.

Diabetes, het gevolg van chronisch hoge bloedsuikerspiegel, veroorzaakt verschillende complicaties in veel delen van het lichaam. Dit is een risicofactor voor hart- en vaatziekten, infecties en het biedt ook een goed klimaat voor de proliferatie van diabetes.

  • Wiebe, Natasha, et al. “Temporal associations among body mass index, fasting insulin, and systemic inflammation: a systematic review and meta-analysis.” JAMA network open 4.3 (2021): e211263-e211263.

Het samengevoegde bewijsmateriaal uit deze meta-analyse suggereert dat verlagingen van de nuchtere insulinespiegels aan gewichtsverlies voorafgaan. Deze bevinding suggereert dat een hoge bloedsuikerspiegel of een andere directe factor de nadelige gevolgen kan veroorzaken die momenteel aan obesitas worden toegeschreven.

  • Wilcox, Gisela. “Insulin and insulin resistance.” Clinical biochemist reviews 26.2 (2005): 19.

Overtuigend bewijs koppelt insulineresistentie aan PCOS.

  • Petersen, Max C., and Gerald I. Shulman. “Mechanisms of insulin action and insulin resistance.” Physiological reviews(2018).

Insulineresistentie is vaak het gevolg van een complex samenspel van reacties op een teveel aan voedingsstoffen. Dit kan leiden tot de ophoping van vetten op ongebruikelijke plaatsen, zoals spieren en lever, wat op zijn beurt de insulineresistentie verergert.

Insulineresistentie kan ernstige gezondheidsproblemen zoals type 2 diabetes veroorzaken.”

  • Sijpkens, Y. W. J., and S. Ramautar. “Koolhydraten, hyperinsulinemie en het insulineresistentie-syndroom.

Door maximaal drie keer per dag te eten kan het lichaam overgaan op vetverbranding, waarbij ketonen als brand- stof worden gebruikt.

  • Quirin, Markus, Julius Kuhl, and Rainer Düsing. “Oxytocin buffers cortisol responses to stress in individuals with impaired emotion regulation abilities.” Psychoneuroendocrinology 36.6 (2011): 898-904.

Oxytocine vergemakkelijkt de stressregulatie.”

  • Kaczmarek, U. (2013). Salivary α-Amylase and Cortisol as Stress Biomarkers – Literature Review α-Amylaza i kortyzol jako biomarkery stresu – przegląd piśmiennictwa. Dental And Medical Problems, 50(3), 271–274. http://www.dmp.umed.wroc.pl/artykuly/DMP_2013503271.pdf”

Cortisol helpt bij het reguleren van je bloedsuikerspiegel zodat je tijdens stress direct energie beschikbaar hebt.”

  • Beheshti, Zahra, et al. “Influence of apple cider vinegar on blood lipids.” Life Science Journal-Acta Zhengzhou University Overseas Edition 9.4 (2012): 2431-2440.

Consumptie van appelciderazijn heeft een gunstig effect op vermindering van schadelijke bloedlipiden en wordt aanbevolen als een eenvoudige en kosteneffectieve behandeling voor een hoge bloedsuikerspiegel.

  • Fahey, Rebecca L. “Health benefits of apple cider vinegar and other common vinegars: A review.” Integrative Medicine Alert20.6 (2017).

Het toevoegen van een kleine hoeveelheid azijn aan je dieet, zoals 1-2 eetlepels ciderazijn per dag, kan al deze voordelen opleveren.

  • Posner, Barry I. “Insulin signalling: the inside story.” Canadian journal of diabetes 41.1 (2017): 108-113.

Wetenschappelijk bewijs toont aan dat herhaalde schommelingen in bloedsuikerspiegels geassocieerd zijn met een verhoogd risico op diabetes, hart- en vaatziekten, obesitas en stemmingsstoornissen zoals depressie.

  • Vasey, Christopher. The water prescription: For health, vitality, and rejuvenation. Simon and Schuster, 2006.

Bronnen wetenschappelijk bewijs voordelen gezonde bloedsuikerspiegel

  • Paula Chandler-Laney et al., “Return of hunger following a relatively high carbohydrate breakfast is associated with earlier recorded glucose peak and nadir,” Appetite 80 (2014): 236-241, https://www.sciencedirect.com/science/article/abs/pii/S0195666314002049.
    Patrick Wyatt et al., “Postprandial glycaemic dips predict appetite and energy intake in healthy individuals,” Nature metabolism 3, no. 4 (2021): 523-529, https://www.nature.com/articles/s42255-021-00383-x.
  • Kathleen Page et al., “Circulating glucose levels modulate neural control of desire for high-calorie foods in humans,” The Journal of clinical investigation 121, no. 10 (2011): 4161-4169, https://www.jci.org/articles/view/57873.
  • Patrick Wyatt et al., “Postprandial glycaemic dips predict appetite and energy intake in healthy individuals,” Nature metabolism 3, no. 4 (2021): 523-529, https://www.nature.com/articles/s42255-021-00383-x.
  • Tanja Taivassalo et al., “The spectrum of exercise tolerance in mitochondrial myopathies: a study of 40 patients,” Brain 126, no. 2 (2003): 413-423, https://pubmed.ncbi.nlm.nih.gov/12538407/.
  • Martin Picard et al., “Mitochondrial allostatic load puts the ‘gluc’ back in glucocorticoids,” Nature Reviews Endocrinology 10, no. 5 (2014): 303-310, https://pubmed.ncbi.nlm.nih.gov/24663223/.
  • Kara L Breymeyer et al., “Subjective mood and energy levels of healthy weight and overweight/obese healthy adults on high-and low-glycemic load experimental diets,” Appetite 107 (2016): 253-259, https://pubmed.ncbi.nlm.nih.gov/27507131/.
  • James Gangwisch et al., “High glycemic index and glycemic load diets as risk factors for insomnia: analyses from the Women’s Health Initiative,” The American journal of clinical nutrition 111, no. 2 (2020): 429-439, https://pubmed.ncbi.nlm.nih.gov/31828298/.
  • R N Aurora et al., “Obstructive Sleep Apnea and Postprandial Glucose Differences in Type 2 Diabetes Mellitus,” In A97. SRN: NEW INSIGHTS INTO THE CARDIOMETABOLIC CONSEQUENCES OF INSUFFICIENT SLEEP, pp. A2525-A2525. American Thoracic Society, 2020, https://www.atsjournals.org/doi/abs/10.1164/ajrccm-conference.2020.201.1_MeetingAbstracts.A2525.
  • Nagham Jafar et al., “The effect of short-term hyperglycemia on the innate immune system,” The American journal of the medical sciences 351, no. 2 (2016): 201-211, https://www.amjmedsci.org/article/S0002-9629(15)00027-0/fulltext.
  • Janna Kiselar et al., “Modification of β-Defensin-2 by dicarbonyls methylglyoxal and glyoxal inhibits antibacterial and chemotactic function in vitro,” PLoS One 10, no. 8 (2015): e0130533, https://journals.plos.org/plosone/article?id=10.1371/journal.pone.0130533.
  • Jiaoyue Zhang et al., “Impaired fasting glucose and diabetes are related to higher risks of complications and mortality among patients with coronavirus disease 2019,” Frontiers in endocrinology 11 (2020): 525, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7365851/.
  • Emmanuelle Logette et al., “A Machine-Generated View of the Role of Blood Glucose Levels in the Severity of COVID-19,” Frontiers in public health 9 (2021): 695139, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8356061/.
  • Francisco Carrasco-Sánchez et al., “Admission hyperglycaemia as a predictor of mortality in patients hospitalized with COVID-19 regardless of diabetes status: data from the Spanish SEMI-COVID-19 Registry,” Annals of medicine 53, no. 1 (2021): 103-116, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7651248.
  • Carol Major et al., “The effects of carbohydrate restriction in patients with diet-controlled gestational diabetes,” Obstetrics & gynecology 91, no. 4 (1998): 600-604, https://www.sciencedirect.com/science/article/abs/pii/S0029784498000039.
  • Robert Moses et al., “Effect of a low-glycemic-index diet during pregnancy on obstetric outcomes,” The American journal of clinical nutrition 84, no. 4 (2006): 807-812, https://www.sciencedirect.com/science/article/pii/S0002916523291017.
  • James F Clapp III, “Maternal carbohydrate intake and pregnancy outcome,” Proceedings of the nutrition society 61, no. 1 (2002): 45-50, https://www.cambridge.org/core/journals/proceedings-of-the-nutrition-society/article/maternal-carbohydrate-intake-and-pregnancy-outcome/28F8E1C5E1460E67F2F1CE0C1D06EE81
  • Rebecca Thurston et al., “Vasomotor symptoms and insulin resistance in the study of women’s health across the nation,” The journal of clinical endocrinology & metabolism 97, no. 10 (2012): 3487-3494, https://pubmed.ncbi.nlm.nih.gov/22851488/.
  • A Fava et al., “Chronic migraine in women is associated with insulin resistance: a cross-sectional study,” European journal of neurology 21, no. 2 (2014): 267-272, https://onlinelibrary.wiley.com/doi/abs/10.1111/ene.12289.
  • James E Gangwisch et al., “High glycemic index and glycemic load diets as risk factors for insomnia: analyses from the Women’s Health Initiative,” The American journal of clinical nutrition 111, no. 2 (2020): 429-439, https://pubmed.ncbi.nlm.nih.gov/31828298/.
  • Rachel Ginieis et al., “The “sweet” effect: comparative assessments of dietary sugars on cognitive performance,” Physiology & behavior 184 (2018): 242-247, https://pubmed.ncbi.nlm.nih.gov/29225094/.
  • A. Nilsson et al., “Effects of differences in postprandial glycaemia on cognitive functions in healthy middle-aged subjects.” European journal of clinical nutrition 63, no. 1 (2009): 113-20, https://www.nature.com/articles/1602900.
  • Hyuck Hoon Kwon et al., “Clinical and histological effect of a low glycaemic load diet in treatment of acne vulgaris in Korean patients: a randomized, controlled trial,” Acta dermato-venereologica 92, no. 3 (2012): 241-246, https://pubmed.ncbi.nlm.nih.gov/22678562/.
  • Robyn N Smith et al., “A low-glycemic-load diet improves symptoms in acne vulgaris patients: a randomized controlled trial,” The American journal of clinical nutrition 86, no. 1 (2007): 107-115, https://pubmed.ncbi.nlm.nih.gov/17616769/.
  • George Suji et al., “Glucose, glycation and aging,” Biogerontology 5, no. 6 (2004): 365-373, https://link.springer.com/article/10.1007/s10522-004-3189-0.
  • Roma Pahwa et al., “Chronic inflammation,” (2018), https://www.ncbi.nlm.nih.gov/books/NBK493173/.
  • Robert A Greenwald et al., “Inhibition of collagen gelation by action of the superoxide radical,” Arthritis & rheumatism: official journal of the American college of rheumatology 22, no. 3 (1979): 251-259, https://pubmed.ncbi.nlm.nih.gov/217393/.
  • Biplab Giri et al., “Chronic hyperglycemia mediated physiological alteration and metabolic distortion leads to organ dysfunction, infection, cancer progression and other pathophysiological consequences: an update on glucose toxicity,” Biomedicine & pharmacotherapy, no. 107 (2018): 306-328, https://www.sciencedirect.com/science/article/abs/pii/S0753332218322406.
  • John Tower, “Programmed cell death in aging,” Ageing research reviews 23 (2015): 90-100, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4480161/
  • Charles Watt et al., “Glycemic variability and CNS inflammation: Reviewing the connection,” Nutrients 12, no. 12 (2020): 3906, https://pubmed.ncbi.nlm.nih.gov/33371247/.
  • F William Danby, “Nutrition and aging skin: sugar and glycation,” Clinics in dermatology 28, no. 4 (2010): 409-411, https://www.sciencedirect.com/science/article/abs/pii/S0738081X10000428.
  • Paraskevi Gkogkolou et al., “Advanced glycation end products: key players in skin aging?” Dermato-endocrinology 4, no. 3 (2012): 259-270, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3583887/.
  • Ashok Katta et al., “Glycation of lens crystalline protein in the pathogenesis of various forms of cataract,” Biomedical Research 20, no. 2 (2009): 119-21, https://www.researchgate.net/profile/Ashok-Katta-3/publication/233419577_Glycation_of_lens_crystalline_protein_in_the_pathogenesis_of_various_forms_of_cataract/links/02e7e531342066c955000000/Glycation-of-lens-crystalline-protein-in-the-pathogenesis-of-various-forms-of-cataract.pdf
  • Suzanne M De la Monte et al., “Alzheimer’s disease is type 3 diabetes—evidence reviewed,” Journal of diabetes science and technology 2, no. 6 (2008): 1101-1113, https://journals.sagepub.com/doi/abs/10.1177/193229680800200619.
  • Robert H, Lustig, Metabolical: The Lure and the Lies of Processed Food, Nutrition, and Modern Medicine (New York: Harper Wave, 2021).
  • Jiyin Zhou et al., “Diabetic cognitive dysfunction: from bench to clinic,” Current medicinal chemistry 27, no. 19 (2020): 3151-3167, https://pubmed.ncbi.nlm.nih.gov/30727866/.
  • Auriel A Willette et al., “Association of insulin resistance with cerebral glucose uptake in late middle–aged adults at risk for Alzheimer disease,” JAMA neurology 72, no. 9 (2015): 1013-1020, https://pubmed.ncbi.nlm.nih.gov/26214150/.
  • Christine M Burns et al., “Higher serum glucose levels are associated with cerebral hypometabolism in Alzheimer regions,” Neurology 80, no. 17 (2013): 1557-1564, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3662330/.
  • Mark A Reger et al., “Effects of β-hydroxybutyrate on cognition in memory-impaired adults,” Neurobiology of aging 25, no. 3 (2004): 311-314, https://www.sciencedirect.com/science/article/abs/pii/S0197458003000873.
  • Dale E Bredesen, “Reversal of cognitive decline: a novel therapeutic program,” Aging (Albany NY) 6, no. 9 (2014): 707-717, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4221920/
  • Amar S Ahmad et al., “Trends in the lifetime risk of developing cancer in Great Britain: comparison of risk for those born from 1930 to 1960.” British journal of cancer 112, no. 5 (2015): 943-947, https://www.nature.com/articles/bjc2014606.
  • Robert H, Lustig, Metabolical: The Lure and the Lies of Processed Food, Nutrition, and Modern Medicine (New York: Harper Wave, 2021).
  • Florian R Greten et al., “Inflammation and cancer: triggers, mechanisms, and consequences,” Immunity 51, no. 1 (2019): 27-41, https://www.sciencedirect.com/science/article/pii/S107476131930295X.
  • Rachel J Perry et al., “Mechanistic links between obesity, insulin, and cancer,” Trends in cancer 6, no. 2 (2020): 75-78, https://www.sciencedirect.com/science/article/abs/pii/S2405803319302614.
  • Tetsuro Tsujimoto et al., “Association between hyperinsulinemia and increased risk of cancer death in nonobese and obese people: A population-based observational study,” International journal of cancer 141, no. 1 (2017): 102-111, https://onlinelibrary.wiley.com/doi/full/10.1002/ijc.30729.
  • Kara L Breymeyer et al., “Subjective mood and energy levels of healthy weight and overweight/obese healthy adults on high-and low-glycemic load experimental diets,” Appetite 107 (2016): 253-259, https://pubmed.ncbi.nlm.nih.gov/27507131/.
  • Rachel A Cheatham et al., “Long-term effects of provided low and high glycemic load low energy diets on mood and cognition,” Physiology & behavior 98, no. 3 (2009): 374-379, https://pubmed.ncbi.nlm.nih.gov/19576915/.
  • Sue Penckofer et al., “Does glycemic variability impact mood and quality of life?” Diabetes technology & therapeutics 14, no. 4 (2012): 303-310, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3317401/.
  • James E Gangwisch et al., “High glycemic index diet as a risk factor for depression: analyses from the Women’s Health Initiative,” The American journal of clinical nutrition 102, no. 2 (2015): 454-463, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4515860/.
  • Fernando F Anhê et al., “Glucose alters the symbiotic relationships between gut microbiota and host physiology,” American journal of physiology-endocrinology and metabolism 318, no. 2 (2020): E111-E116, https://pubmed.ncbi.nlm.nih.gov/31794261/.
  • Robert H, Lustig, Metabolical: The Lure and the Lies of Processed Food, Nutrition, and Modern Medicine (New York: Harper Wave, 2021).
  • William S Yancy et al., “Improvements of gastroesophageal reflux disease after initiation of a low-carbohydrate diet: Five brief case reports,” Alternative therapies in health and medicine 7, no. 6 (2001): 116-119, https://pubmed.ncbi.nlm.nih.gov/11712463/.
  • Koichi Node et al., “Postprandial hyperglycemia as an etiological factor in vascular failure,” Cardiovascular Diabetology 8 (2009): 23, https://pubmed.ncbi.nlm.nih.gov/19402896/.
  • Antonio Ceriello et al., “Oscillating glucose is more deleterious to endothelial function and oxidative stress than mean glucose in normal and type 2 diabetic patients,” Diabetes 57, no. 5 (2008): 1349-1354, https://pubmed.ncbi.nlm.nih.gov/18299315/.
  • Michelle C Flynn et al., “Transient intermittent hyperglycemia accelerates atherosclerosis by promoting myelopoiesis,” Circulation research 127, no. 7 (2020): 877-892, https://www.ahajournals.org/doi/full/10.1161/CIRCRESAHA.120.316653.
  • E Succurro et al., “Elevated one-hour post-load plasma glucose levels identifies subjects with normal glucose tolerance but early carotid atherosclerosis,” Atherosclerosis 207, no. 1 (2009): 245-249, https://www.sciencedirect.com/science/article/abs/pii/S0021915009002718.
  • Tetsurou Sakumoto et al., “Insulin resistance/hyperinsulinemia and reproductive disorders in infertile women,” Reproductive medicine and biology 9, no. 4 (2010): 185-190, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5904600/.
  • LaTasha B Craig et al., “Increased prevalence of insulin resistance in women with a history of recurrent pregnancy loss,” Fertility and sterility 78, no. 3 (2002): 487-490, https://www.sciencedirect.com/science/article/abs/pii/S0015028202032478.
  • Nelly Pitteloud et al., “Increasing insulin resistance is associated with a decrease in Leydig cell testosterone secretion in men,” The journal of clinical endocrinology & metabolism 90, no. 5 (2005): 2636-2641, https://academic.oup.com/jcem/article/90/5/2636/2836773.
  • Jorge E Chavarro et al., “A prospective study of dietary carbohydrate quantity and quality in relation to risk of ovulatory infertility,” European journal of clinical nutrition 63, no. 1 (2009): 78-86, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3066074/.
  • Zeeshan Anwar et al., “Erectile dysfunction: An underestimated presentation in patients with diabetes mellitus,” Indian journal of psychological medicine 39, no. 5 (2017): 600-604, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC5688886/.
  • Fengjuan Yao et al., “Erectile dysfunction may be the first clinical sign of insulin resistance and endothelial dysfunction in young men,” Clinical research in cardiology 102, no. 9 (2013): 645-651, https://link.springer.com/article/10.1007/s00392-013-0577-y.
  • John E Nestler et al., “Insulin stimulates testosterone biosynthesis by human thecal cells from women with polycystic ovary syndrome by activating its own receptor and using inositolglycan mediators as the signal transduction system,” The Journal of clinical endocrinology & metabolism 83, no. 6 (1998): 2001-2005, https://academic.oup.com/jcem/article/83/6/2001/2865383.
  • Benjamin Bikman, Why We Get Sick: The Hidden Epidemic at the Root of Most Chronic Disease and How to Fight It (New York: BenBella, 2020).
  • John C Mavropoulos et al., “The effects of a low-carbohydrate, ketogenic diet on the polycystic ovary syndrome: a pilot study,” Nutrition & metabolism 2 (2005): 35, https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1334192/.
  • Zobair M Younossi et al., “Global epidemiology of nonalcoholic fatty liver disease—meta analytic assessment of prevalence, incidence, and outcomes,” Hepatology 64, no. 1 (2016): 73-84, https://aasldpubs.onlinelibrary.wiley.com/doi/full/10.1002/hep.28431.
  • Ruth C R Meex et al., “Hepatokines: linking nonalcoholic fatty liver disease and insulin resistance.” Nature Reviews Endocrinology 13, no. 9 (2017): 509-520, https://www.nature.com/articles/nrendo.2017.56.
  • Marc Y Donath et al., “Type 2 diabetes as an inflammatory disease,” Nature reviews immunology 11, no. 2 (2011): 98-107, https://pubmed.ncbi.nlm.nih.gov/21233852/.
  • Joshua Z Goldenberg et al., “Efficacy and safety of low and very low carbohydrate diets for type 2 diabetes remission: systematic review and meta-analysis of published and unpublished randomized trial data,” bmj 372 (2021): m4743, https://www.bmj.com/content/372/bmj.m4743.
  • William S Yancy et al., “A low-carbohydrate, ketogenic diet to treat type 2 diabetes,” Nutrition & metabolism 2 (2005): 34, https://link.springer.com/article/10.1186/1743-7075-2-34.
  • Alison B Evert et al., “Nutrition therapy for adults with diabetes or prediabetes: a consensus report,” Diabetes care 42, no. 5 (2019): 731-754, https://pubmed.ncbi.nlm.nih.gov/31000505/.

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